Constitutively expressed by a wide spectrum of cells and tissues, including monocytes and macrophages, MIF is quickly introduced immediately after publicity to microbial solutions and pro-inflammatory mediators, and in response to stress.
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even so, very little is thought in regards to the relevance of such putative DNA-binding web-sites in the regulation of expression in the human MIF gene. Two polymorphisms on the human MIF gene (arrows) — a CATT-tetranucleotide sequence recurring 5 to eight situations at situation −794 in addition to a G-to-C one nucleotide polymorphism (SNP) at position −173 — happen to be associated with the severity of rheumatoid arthritis and systemic-onset juvenile idiopathic arthritis.
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